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- New way to monitor and prevent nerve cell deterioration after TBIon April 13, 2021 at 4:43 pm
Researchers have discovered a new way to prevent brain nerve cells from deteriorating after injury, which also revealed a potential mechanistic link between TBI and AD. Their discovery also yielded a new blood biomarker of nerve cell degeneration after injury, which is significant because there is an urgent need for mechanism-based blood biomarkers that can diagnose TBI and stage its severity.
- Profound loss of pleasure related to early-onset dementiaon April 12, 2021 at 2:19 pm
Loss of the ability to experience pleasure - or anhedonia - has been revealed as a key feature in frontotemporal dementia, in contrast to Alzheimer's disease. The findings from brain scans, believed to be a first, show grey matter deterioration in the so-called pleasure system of the brain - these regions were distinct from those implicated in depression or apathy, suggesting a possible treatment target for the early-onset dementia that affects people from 40-65 years.
- Imbalance in gum bacteria linked to Alzheimer's disease biomarkeron April 12, 2021 at 12:45 pm
Older adults with more harmful than healthy bacteria in their gums are more likely to have evidence for amyloid beta -- a key biomarker for Alzheimer's disease -- in their cerebrospinal fluid, according to new research. However, this imbalance in oral bacteria was not associated with another Alzheimer's biomarker called tau.
- Imbalance in gum bacteria linked to Alzheimer's disease biomarkeron April 12, 2021 at 12:45 pm
Older adults with more harmful than healthy bacteria in their gums are more likely to have evidence for amyloid beta -- a key biomarker for Alzheimer's disease -- in their cerebrospinal fluid, according to new research. However, this imbalance in oral bacteria was not associated with another Alzheimer's biomarker called tau.
- Aluminum is intricately associated with the neuropathology of familial Alzheimer's diseaseon April 9, 2021 at 4:47 pm
A new study continues to support a growing body of evidence that aluminum contributes to the pathogenesis of Alzheimer's disease (AD). Researchers found aluminum co-located with phosphorylated tau protein, which is an early initiator of AD.